急性肾损伤常常伴随一场修复以及肾脏纤维化,但是介导AKI这些急性和慢性后果的分子机制目前仍不完全清楚。因此,本书的研究应用表皮生长因子受体基因缺陷小数以及它们的同窝野生型小数,建立单侧肾缺血在灌注损伤动物模型,来检测EGFR在这些过程中的作用。
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急性肾损伤常常伴随一场修复以及肾脏纤维化,但是介导AKI这些急性和慢性后果的分子机制目前仍不完全清楚。因此,本书的研究应用表皮生长因子受体基因缺陷小数以及它们的同窝野生型小数,建立单侧肾缺血在灌注损伤动物模型,来检测EGFR在这些过程中的作用。
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